Download PDF by M. Pfreundschuh, H. Shiku, T. Takahashi, R. Ueda (auth.),: Cancer Chemo- and Immunopharmacology: 2: Immunopharmacology,

By M. Pfreundschuh, H. Shiku, T. Takahashi, R. Ueda (auth.), Professor Georges Mathé, Professor Franco M. Muggia (eds.)

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Extra info for Cancer Chemo- and Immunopharmacology: 2: Immunopharmacology, Relations, and General Problems

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Cell Immunol 45: 36-48 3 Caraux J, Thierry C, Serrou B (1979) Human autologous rosettes. II. Prognostic significance of variations in autologous rosette forming cells in the peripheral blood of cancer patients. J Natl Cancer Inst 63: 593-601 4 Gaud L, Caraux J, Thierry C, Pujol H, Serrou B (1978) Modifications of the immune status induced in malignant melanoma patients by chemo-immunotherapy and immunotherapy. In: Serrou B, Rosenfeld C (eds) Human lymphocyte differentiation. Its application to cancer.

In contrast, evaluation of total T-Iymphocyte numbers by the E-rosette assay does not discriminate between these two states [3]. Conclusions Overall, these results point out the complexity involved with investigation of fine modifications of the immune response. Two aspects should be retained: a specific Immune Imbalance in Cancer Patients 45 ABSOLlITE NLtIlER OF A-RFC PER ~ OF BLOOD A-RFC/MM 3 500 ltOO 200 100 Fig. 2. ,7 I£IIISS [(II 273 RELAPSE 32 aspect which still remains problematic, at least for solid tumors, and a nonspecific aspect where advances in the purification and characterization of subpopulations and an understanding of the intimate mechanisms of the immune response give rise to a new approach opening into some practical perspectives.

Cancer Immunol Immunother 4: 239- 245 Suppressor Cells in Hodgkin's Disease and Lung Carcinoma R. Ruchet, Y. Thomas, and D. Grandjon Summary Lymphocyte response to PHA was depressed in 9 of 14 cases of Hodgkin's disease (HD) and in 4 of 14 cases of lung carcinoma (LC). This depression was caused in one-third of the HD cases and in one-half of the LC cases by an excess of prostaglandin synthesis. Introduction The mechanisms underlying the depression of cell-mediated immunity (CMI) in malignant diseases are not very well understood.

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