By Michelle Bowman-Howard MD
Anesthesia evaluation, moment Edition is the right learn relief for either the written and the oral board examinations in anesthesiology. the 1st part offers board-format questions in all of the significant components of anesthesiology, together with body structure, pharmacology, local anesthesia, ache administration, and significant organ structures. the second one part presents bulleted, outline-format solutions that overview the data had to solution the query appropriately. The 3rd part comprises instances to additional organize readers for oral boards.
This variation contains completely up to date info, rather on airway administration, pharmacology, sufferer safeguard, and caliber coverage. Questions and solutions comprise references to the most recent variants of normal anesthesiology textbooks.
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What is the treatment? What do you do for tachycardia-induced AV block? 2. What are some of the antecedent factors for atrial fibrillation? What is the treatment? 3. What are the two kinds of atrial flutter? What are the two most common causes of atrial flutter? What are the treatments? 4. Where do you see the P wave on the ECG in AV nodal reentrant tachycardia? What is the treatment of choice? How else can it be treated? 5. What is Wolff-Parkinson-White (WPW) syndrome? What is the circuit? What are the characteristics seen on ECG?
5. 6. 7. 8. What are the preoperative findings that are suggestive of ventricular dysfunction? Which drugs should be continued preoperatively? What is a common premedication given for CPB? Which monitors are used for CPB? After CPB, is there a difference between central and peripheral blood pressures? If so, why? What is the main benefit of using inhalation anesthetics in cardiac surgery? Name four problems associated with a high-dose opioid anesthetic technique. When should N2 O not be used in cardiac surgery?
5. 6. 7. 8. What are the signs/symptoms of CNS toxicity from LAs? What are the signs of CV toxicity from LAs? What is the treatment for CNS toxicity of LAs? What factors increase CNS toxicity? What is it caused by? What decreases systemic toxicity? What are the systemic effects of intra-arterial injection of LAs? What are the LA effects on the CV system? What is the mechanism of action? Why are long-acting amides (such as bupivicaine) so dangerous? What is the difference between bupivicaine and ropivicaine in this regard?